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Robert S. B. Clark

Researcher at University of Pittsburgh

Publications -  298
Citations -  25842

Robert S. B. Clark is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Traumatic brain injury & Cerebral blood flow. The author has an hindex of 74, co-authored 279 publications receiving 23107 citations. Previous affiliations of Robert S. B. Clark include Joint Base San Antonio & Boston Children's Hospital.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
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Unexpected Increased Mortality After Implementation of a Commercially Sold Computerized Physician Order Entry System

Yong Y. Han, +8 more
- 01 Dec 2005 - 
TL;DR: An unexpected increase in mortality coincident with CPOE implementation is observed, which suggests that when implementing C POE systems, institutions should continue to evaluate mortality effects, in addition to medication error rates, for children who are dependent on time-sensitive therapies.
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The far-reaching scope of neuroinflammation after traumatic brain injury

Dennis W. Simon, +5 more
- 01 Mar 2017 - 
TL;DR: A new framework of targeted immunomodulation after TBI is proposed that incorporates factors such as the time from injury, mechanism of injury, and secondary insults in considering potential treatment options and highlights findings that could offer novel therapeutic targets for translational and clinical research.
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Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury.

Milos D. Ikonomovic, +9 more
- 01 Nov 2004 - 
TL;DR: The results demonstrate a differential distribution and course of intra- and extra-cellular AD-like changes during the acute phase following severe TBI in humans, and provide insight into the molecular mechanisms that initiate these pathological cascades very early during severe brain injury.