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Raymond Chuen-Chung Chang

Researcher at University of Hong Kong

Publications -  183
Citations -  18689

Raymond Chuen-Chung Chang is an academic researcher from University of Hong Kong. The author has contributed to research in topics: Neuroprotection & Neurotoxicity. The author has an hindex of 53, co-authored 175 publications receiving 15898 citations. Previous affiliations of Raymond Chuen-Chung Chang include National Institutes of Health & The Chinese University of Hong Kong.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Effects of all-trans-retinoic acid on human SH-SY5Y neuroblastoma as in vitro model in neurotoxicity research.

TL;DR: It is hypothesized that modulation of signaling pathways and neuronal properties during RA-mediated differentiation in SH-SY5Y cells can affect their susceptibility to neurotoxins, and undifferentiated SH- SY5Y is more appropriate for studying neurotoxicity or neuroprotection in experimental Parkinson's disease research.
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Use of Anti-aging Herbal Medicine, Lycium barbarum , Against Aging-associated Diseases. What Do We Know So Far?

TL;DR: Lycium barbarum can protect neurons against β-amyloid peptide toxicity in neuronal cell cultures, and retinal ganglion cells in an experimental model of glaucoma, and its neuroprotective effects to counter neuronal loss in neurodegenerative diseases are demonstrated.