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Ciro Isidoro

Researcher at University of Eastern Piedmont

Publications -  180
Citations -  16769

Ciro Isidoro is an academic researcher from University of Eastern Piedmont. The author has contributed to research in topics: Autophagy & Cathepsin D. The author has an hindex of 42, co-authored 161 publications receiving 13684 citations. Previous affiliations of Ciro Isidoro include Health Science University & University of Münster.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Lithium delays progression of amyotrophic lateral sclerosis.

TL;DR: It is found that daily doses of lithium, leading to plasma levels ranging from 0.4 to 0.8 mEq/liter, delay disease progression in human patients affected by ALS, and a marked neuroprotection by lithium is found, which delayed disease onset and duration and augmented the life span.
Journal ArticleDOI

Autophagic-lysosomal perturbation enhances tau aggregation in transfectants with induced wild-type tau expression.

TL;DR: The results suggest that the autophagic‐lysosomal system plays a role in the clearance of t Tau, and that dysfunction of this system results in the formation of tau oligomers and insoluble aggregates.