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Guillermo Mariño

Researcher at University of Oviedo

Publications -  82
Citations -  24561

Guillermo Mariño is an academic researcher from University of Oviedo. The author has contributed to research in topics: Autophagy & Progeria. The author has an hindex of 45, co-authored 75 publications receiving 20454 citations. Previous affiliations of Guillermo Mariño include French Institute of Health and Medical Research & Paris Descartes University.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Autophagy and the Integrated Stress Response

TL;DR: Autophagy is a cell biological process that is a central component of the integrated stress response and can be integrated with other cellular stress responses through parallel stimulation of autophagy and other stress responses by specific stress stimuli.
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Autophagy and Aging

TL;DR: The probable cause and effect relationship between perturbed autophagy and aging is discussed, as well as possible molecular mechanisms that may mediate the anti-aging effects of Autophagy.
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Self-consumption: the interplay of autophagy and apoptosis

TL;DR: The dialogue between autophagy and cell death pathways influences the normal clearance of dying cells, as well as immune recognition of dead cell antigens, and the disruption of the relationship between autphagy and apoptosis has important pathophysiological consequences.