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Peter G.H. Clarke

Researcher at University of Lausanne

Publications -  88
Citations -  18960

Peter G.H. Clarke is an academic researcher from University of Lausanne. The author has contributed to research in topics: Programmed cell death & Neuroprotection. The author has an hindex of 38, co-authored 88 publications receiving 17231 citations.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
Journal ArticleDOI

Developmental cell death: morphological diversity and multiple mechanisms.

TL;DR: This review concentrates on an aspect of developmental cell death that has tended to be neglected, the manner in which the cells are dismantled, which appears to be achieved primarily by heterophagy, by autophagy and by non-lysosomal degradation, respectively.
Journal ArticleDOI

A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemia.

TL;DR: A cell-penetrating, protease-resistant peptide that blocks the access of JNK to many of its targets is evaluated and is found to be a promising neuroprotective agent for stroke.