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Leonidas C. Platanias

Researcher at Northwestern University

Publications -  394
Citations -  30356

Leonidas C. Platanias is an academic researcher from Northwestern University. The author has contributed to research in topics: Signal transduction & PI3K/AKT/mTOR pathway. The author has an hindex of 77, co-authored 353 publications receiving 27017 citations. Previous affiliations of Leonidas C. Platanias include Veterans Health Administration & Loyola University Chicago.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Mechanisms of type-I- and type-II-interferon-mediated signalling.

TL;DR: It is anticipated that an increased understanding of the contributions of these recently identified pathways will advance current thinking about how interferons work.
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Map kinase signaling pathways and hematologic malignancies

TL;DR: The involvement of various Map kinase pathways in the pathophysiology of hematologic malignances is summarized and the clinical implications of the recent advances in the field are discussed.
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Direct association with and dephosphorylation of jak2 kinase by the sh2-domain-containing protein tyrosine phosphatase shp-1

TL;DR: The interactions of SHP-1 with two tyrosine kinases activated during engagement of the erythropoietin receptor, the Janus family kinase Jak-2 and the c-fps/fes kinase are examined to suggest the existence of similar mechanisms for other members of the Jak family of kinases.