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Eitan Shaulian

Researcher at Hebrew University of Jerusalem

Publications -  23
Citations -  10415

Eitan Shaulian is an academic researcher from Hebrew University of Jerusalem. The author has contributed to research in topics: Transcription factor & JUNB. The author has an hindex of 19, co-authored 22 publications receiving 9072 citations. Previous affiliations of Eitan Shaulian include Weizmann Institute of Science & University of California, San Diego.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

AP-1 as a regulator of cell life and death

Eitan Shaulian, +1 more
- 01 May 2002 - 
TL;DR: Interestingly, the growth-promoting activity of c-Jun is mediated by repression of tumour suppressors, as well as upregulation of positive cell cycle regulators, whereas JunB has the converse effect.
Journal ArticleDOI

AP-1 in cell proliferation and survival.

Eitan Shaulian, +1 more
- 30 Apr 2001 - 
TL;DR: Amongst the Jun proteins, c-Jun is unique in its ability to positively regulate cell proliferation through the repression of tumor suppressor gene expression and function, and induction of cyclin D1 transcription.
Journal ArticleDOI

AP-1 — The Jun proteins: Oncogenes or tumor suppressors in disguise?

Eitan Shaulian
- 01 Jun 2010 - 
TL;DR: The current knowledge about the roles of Jun proteins in human neoplasia is described and pathological examples demonstrating that the initial dogma has to be reexamined are focused on.
Journal ArticleDOI

The mammalian UV response: c-Jun induction is required for exit from p53-imposed growth arrest.

Eitan Shaulian, +5 more
- 08 Dec 2000 - 
TL;DR: An explanation for the role of c-Jun in the UV response of mouse fibroblasts is provided, which is necessary for cell-cycle reentry of UV-irradiated cells, but does not participate in the response to ionizing radiation.