scispace - formally typeset
M

Matthias Nauck

Researcher at Greifswald University Hospital

Publications -  615
Citations -  49935

Matthias Nauck is an academic researcher from Greifswald University Hospital. The author has contributed to research in topics: Population & Study of Health in Pomerania. The author has an hindex of 91, co-authored 544 publications receiving 41655 citations. Previous affiliations of Matthias Nauck include University of Greifswald & University of Freiburg.

Papers
More filters
Journal ArticleDOI

Associations of autozygosity with a broad range of human phenotypes

David W. Clark, +496 more
TL;DR: In this paper, the authors used genomic inbreeding coefficients (FROH) for >1.4 million individuals and found that FROH is significantly associated with apparently deleterious changes in 32 out of 100 traits analysed.
Journal ArticleDOI

35th Annual Meeting of the European Association for the Study of Diabetes : Brussels, Belgium, 28 September-2 October 1999.

Arne Melander, +4778 more
- 01 Aug 1999 - 
Journal ArticleDOI

Women are periodontally healthier than men, but why don't they have more teeth than men?

TL;DR: The apparent paradox of having fewer teeth despite better periodontal health in women compared with men is related to an increased bone turnover rate and socioeconomic conditions such as low education and low social status.
Journal ArticleDOI

Prospective association of low total testosterone concentrations with an adverse lipid profile and increased incident dyslipidemia.

TL;DR: Low total testosterone concentrations are prospectively associated with an adverse lipid profile and increased risk of incident dyslipidemia, which may contribute to an explanation for the higher cardiovascular disease risk in men with lower totalosterone concentrations.
Journal ArticleDOI

Serum prolactin concentrations as risk factor of metabolic syndrome or type 2 diabetes

TL;DR: The present study is the first large population-based study reporting a cross-sectional inverse association between PRL and prevalent T2DM in both genders, and the absent longitudinal associations do not support a causal role of PRL as a risk factor of incident MetS or T2 DM.