scispace - formally typeset
E

Eduardo Couve

Researcher at Valparaiso University

Publications -  39
Citations -  10860

Eduardo Couve is an academic researcher from Valparaiso University. The author has contributed to research in topics: Myelin & Odontoblast. The author has an hindex of 21, co-authored 38 publications receiving 9334 citations. Previous affiliations of Eduardo Couve include University of Santo Tomas & University of Valparaíso.

Papers
More filters
Journal ArticleDOI

Aminochrome induces disruption of actin, alpha-, and beta-tubulin cytoskeleton networks in substantia-nigra-derived cell line.

Irmgard Paris, +7 more
- 20 Jan 2010 - 
TL;DR: The results suggest that aminochrome one-electron metabolism induces the disruption of the normal morphology of actin, alpha-, and beta-tubulin in the cytoskeleton, and that DT-diaphorase prevents these effects.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

DT-Diaphorase Prevents Aminochrome-Induced Alpha-Synuclein Oligomer Formation and Neurotoxicity

Patricia Muñoz, +6 more
- 01 May 2015 - 
TL;DR: It is found that DT-diaphorase (NQO1) prevents the formation of SNCA oligomers in the presence of aminochrome determined by Western blot, transmission electron microscopy, circular dichroism, and thioflavin T fluorescence, suggesting a protective role of N QO1 by preventing the formation in dopaminergic neurons.
Journal ArticleDOI

Copper dopamine complex induces mitochondrial autophagy preceding caspase-independent apoptotic cell death

Irmgard Paris, +5 more
- 15 May 2009 - 
TL;DR: The results suggest that the copper·dopamine complex induces mitochondrial autophagy followed by caspase-3-independent apoptotic cell death in RCSN-3 cells, which is one of the major neurological symptoms in Wilson disease.
Journal ArticleDOI

Dynamin-2 regulates fusion pore expansion and quantal release through a mechanism that involves actin dynamics in neuroendocrine chromaffin cells.

Arlek M. González-Jamett, +13 more
- 05 Aug 2013 - 
TL;DR: A model in which dynamin-2 directs actin polymerization at the exocytosis site where both, in concert, adjust the hormone quantal release to efficiently respond to physiological demands is supported.