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J. David Beckham

Researcher at University of Colorado Denver

Publications -  71
Citations -  6954

J. David Beckham is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Medicine & Zika virus. The author has an hindex of 21, co-authored 57 publications receiving 6068 citations. Previous affiliations of J. David Beckham include Baylor College of Medicine & University of Colorado Boulder.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Zika virus produces noncoding RNAs using a multi-pseudoknot structure that confounds a cellular exonuclease

Benjamin M. Akiyama, +10 more
- 02 Dec 2016 - 
TL;DR: The authors solved the structure of one of ZIKV's sfRNAs by x-ray crystallography and found that the multi-pseudoknot structure that it adopts underlies its exonuclease resistance, clarifying the mechanism of exonuclelease resistance and identifies features that may modulate function in diverse flaviviruses.
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Respiratory viral infections in patients with chronic, obstructive pulmonary disease

J. David Beckham, +6 more
- 01 May 2005 - 
TL;DR: Respiratory viral infections are commonly associated with acute respiratory illness in COPD patients, and the use of RT-PCR assays significantly increases the ability to diagnose these infections.
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Alpha-Synuclein Expression Restricts RNA Viral Infections in the Brain

Erica L. Beatman, +6 more
- 15 Mar 2016 - 
TL;DR: It is discovered that native neuronal expression of alpha-synuclein inhibited viral infection in the central nervous system, and this work proposes a model in which virus-induced Asyn localizes to ER-derived membranes, modulates virus- induced ER stress signaling, and inhibits viral replication, growth, and injury in the CNS.
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Zika virus: An emergent neuropathological agent.

Martyn K. White, +4 more
- 01 Oct 2016 - 
TL;DR: The rapid advance of the virus in the Americas and its likely association with microcephaly and Guillain–Barré syndrome make Zika an urgent public health concern.