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Emilio Clementi

Researcher at University of Milan

Publications -  441
Citations -  22132

Emilio Clementi is an academic researcher from University of Milan. The author has contributed to research in topics: Nitric oxide & Medicine. The author has an hindex of 57, co-authored 411 publications receiving 19190 citations. Previous affiliations of Emilio Clementi include University of Calabria & Laboratory of Molecular Biology.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mitochondrial biogenesis in mammals: The role of endogenous nitric oxide

Enzo Nisoli, +10 more
- 07 Feb 2003 - 
TL;DR: The mitochondrial biogenesis induced by exposure to cold was markedly reduced in brown adipose tissue of endothelial nitric oxide synthase null-mutant (eNOS–/–) mice, which had a reduced metabolic rate and accelerated weight gain as compared to wild-type mice.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Calorie Restriction Promotes Mitochondrial Biogenesis by Inducing the Expression of eNOS

Enzo Nisoli, +12 more
- 14 Oct 2005 - 
TL;DR: Nitric oxide plays a fundamental role in the processes induced by calorie restriction and may be involved in the extension of life span in mammals.
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Acid sphingomyelinase activity triggers microparticle release from glial cells.

Fabio Bianco, +11 more
- 22 Apr 2009 - 
TL;DR: The results represent the first demonstration that activation of acid sphingomyelinase is necessary and sufficient for microparticle release from glial cells and define key molecular effectors of microparticles formation and IL‐1β release, thus, opening new strategies for the treatment of neuroinflammatory diseases.