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Thomas Vaccari

Researcher at University of Milan

Publications -  71
Citations -  10165

Thomas Vaccari is an academic researcher from University of Milan. The author has contributed to research in topics: Autophagy & ESCRT. The author has an hindex of 27, co-authored 65 publications receiving 8456 citations. Previous affiliations of Thomas Vaccari include University of California, Berkeley & University of Trieste.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Biologic response of B lymphoma cells to anti-CD20 monoclonal antibody rituximab in vitro: CD55 and CD59 regulate complement-mediated cell lysis

Josée Golay, +15 more
- 15 Jun 2000 - 
TL;DR: It is concluded that CDC and ADCC are major mechanisms of action of rituximab on B-cell lymphomas and that a heterogeneous susceptibility of different lymphoma cells to complement may be at least in part responsible for the heterogeneity of the response of different patients to ritUXimab in vivo.
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The Drosophila Tumor Suppressor vps25 Prevents Nonautonomous Overproliferation by Regulating Notch Trafficking

Thomas Vaccari, +1 more
- 01 Nov 2005 - 
TL;DR: Vps25, a component of the ESCRT machinery that regulates endocytic sorting of signaling receptors, is identified as an unconventional type of Drosophila tumor suppressor.
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ESCRTs and Fab1 Regulate Distinct Steps of Autophagy

Tor Erik Rusten, +10 more
- 23 Oct 2007 - 
TL;DR: This study links ESCRT function with autophagy and aggregate-induced neurodegeneration, thereby providing a plausible explanation for the fact that ESCRT mutations are involved in inherited neurodegenersative disease in humans.