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Cinzia Fabrizi

Researcher at Sapienza University of Rome

Publications -  51
Citations -  6309

Cinzia Fabrizi is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Microglia & Receptor. The author has an hindex of 19, co-authored 50 publications receiving 5804 citations. Previous affiliations of Cinzia Fabrizi include University of Salerno & Catholic University of the Sacred Heart.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Involvement of the intracellular ion channel CLIC1 in microglia-mediated beta-amyloid-induced neurotoxicity.

Gaia Novarino, +11 more
- 09 Jun 2004 - 
TL;DR: Reducing CLIC1 chloride conductance by a specific blocker prevents neuronal apoptosis in neurons cocultured with Aβ-treated microglia and prevents TNF-α release induced by Aβ stimulation, providing a direct link between A β-induced microglial activation andCLIC1 functional expression.
Journal ArticleDOI

Activation of microglial cells by PrP and β-amyloid fragments raises intracellular calcium through L-type voltage sensitive calcium channels

Valerio Silei, +6 more
- 06 Feb 1999 - 
TL;DR: Results show that both peptides are able to induce microglial activation and to elicit an increase in [Ca2+]i levels in cells loaded with calcium-green 1, thus indicating a transmembrane calcium influx through these channels.
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S100B protects LAN-5 neuroblastoma cells against Abeta amyloid-induced neurotoxicity via RAGE engagement at low doses but increases Abeta amyloid neurotoxicity at high doses.

Rita Businaro, +6 more
- 01 Apr 2006 - 
TL;DR: The data suggest that at nanomolar doses S100B counteracts Aβ peptide neurotoxicity in a RAGE‐mediated manner, however, at micromolar dosesS100B is toxic to LAN‐5 cells and its toxicity adds to that of the A β peptide, suggesting that additional molecular mechanisms may be involved in the neurotoxic process.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 -