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Skye C. McIver

Researcher at University of Wisconsin-Madison

Publications -  9
Citations -  5480

Skye C. McIver is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Cellular differentiation & Erythroid Precursor Cells. The author has an hindex of 8, co-authored 9 publications receiving 5018 citations. Previous affiliations of Skye C. McIver include Wisconsin Alumni Research Foundation.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

The exosome complex establishes a barricade to erythroid maturation.

Skye C. McIver, +11 more
- 02 Oct 2014 - 
TL;DR: These results demonstrate a new mode of controlling erythropoiesis in which multiple components of the exosome complex are endogenous suppressors of the erythroid developmental program.
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

Integrating Enhancer Mechanisms to Establish a Hierarchical Blood Development Program.

Charu Mehta, +7 more
- 19 Sep 2017 - 
TL;DR: The mechanisms were dissected using -77;+9.5 compound heterozygous (CH) mice and found that the enhancer circuits controlled signaling pathways that orchestrate a GATA factor-dependent blood development program.