scispace - formally typeset
D

Damián Gatica

Researcher at University of Michigan

Publications -  25
Citations -  8262

Damián Gatica is an academic researcher from University of Michigan. The author has contributed to research in topics: Autophagy & Unfolded protein response. The author has an hindex of 13, co-authored 24 publications receiving 6457 citations. Previous affiliations of Damián Gatica include University of Chile & Life Sciences Institute.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Cargo recognition and degradation by selective autophagy

TL;DR: Different types of selective autophagy are discussed, emphasizing the role of ligand receptors and scaffold proteins in providing cargo specificity, and unanswered questions in the field are highlighted.
Book ChapterDOI

Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integration.

TL;DR: In this article, the distinct functions of the endoplasmic reticulum (ER) and unfolded protein response (UPR) from a metabolic point of view are reviewed, highlighting their association with prevalent pathologies.
Journal ArticleDOI

Endoplasmic reticulum: ER stress regulates mitochondrial bioenergetics

TL;DR: How ER influences mitochondrial function through mechanisms involving calcium transfer, thereby facilitating cellular adaptation and how dysregulation of ER-mitochondrial calcium homeostasis during prolonged ER stress is emerging as a novel mechanism implicated in the onset of metabolic disorders are highlighted.