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Karin Nowikovsky

Researcher at Medical University of Vienna

Publications -  34
Citations -  6741

Karin Nowikovsky is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Mitochondrion & LETM1. The author has an hindex of 17, co-authored 27 publications receiving 6091 citations. Previous affiliations of Karin Nowikovsky include University of Veterinary Medicine Vienna & Max F. Perutz Laboratories.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mdm38 protein depletion causes loss of mitochondrial K+/H+ exchange activity, osmotic swelling and mitophagy.

Karin Nowikovsky, +3 more
- 01 Jun 2007 - 
TL;DR: It is shown that osmotic swelling of mitochondria triggers selective mitochondrial autophagy or mitophagy, and loss of K+/H+ exchange activity and mitochondrial swelling are early events, associated with a reduction in membrane potential and fragmentation of the mitochondrial reticulum.
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Virtual reality for freely moving animals

John R. Stowers, +14 more
- 21 Aug 2017 - 
TL;DR: By photorealistically mimicking zebrafish, the development of FreemoVR, a VR system for freely moving animals, is reported and it is discovered that effective social influence depends on a prospective leader balancing its internally preferred directional choice with social interaction.
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Muscle-Specific Loss of Apoptosis-Inducing Factor Leads to Mitochondrial Dysfunction, Skeletal Muscle Atrophy, and Dilated Cardiomyopathy

Nicholas Joza, +16 more
- 01 Dec 2005 - 
TL;DR: Tissue-specific deletion of Aif is reported in the mouse to provide genetic proof that the previously defined cell death promoter AIF has a second essential function in mitochondrial respiration and aerobic energy metabolism required for normal heart function and skeletal muscle homeostasis.
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The LETM1/YOL027 Gene Family Encodes a Factor of the Mitochondrial K+ Homeostasis with a Potential Role in the Wolf-Hirschhorn Syndrome

Karin Nowikovsky, +7 more
- 16 Jul 2004 - 
TL;DR: YOL027/LETM1 are the first genes shown to encode factors involved in both K+ homeostasis and organelle volume control, indicating a functional conservation of LetM1/Yol027 proteins from yeast to man.