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Reinhard Dechant

Researcher at ETH Zurich

Publications -  28
Citations -  6832

Reinhard Dechant is an academic researcher from ETH Zurich. The author has contributed to research in topics: Cell growth & Nutrient sensing. The author has an hindex of 16, co-authored 28 publications receiving 6157 citations. Previous affiliations of Reinhard Dechant include Research Institute of Molecular Pathology & Max F. Perutz Laboratories.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Cytosolic pH is a second messenger for glucose and regulates the PKA pathway through V-ATPase.

Reinhard Dechant, +5 more
- 04 Aug 2010 - 
TL;DR: This work identifies cytosolic pH as a second messenger for glucose that mediates activation of the PKA pathway in yeast and identifies the vacuolar ATPase (V‐ATPase), a proton pump required for the acidification of vacuoles, as a sensor of cytosol pH.
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Mass spectrometry-based metabolomics of single yeast cells

Alfredo J. Ibáñez, +8 more
- 28 May 2013 - 
TL;DR: Correlation plots between metabolites from the glycolytic pathway, as well as with the observed ATP/ADP ratio as a measure of cellular energy charge, give biological insight not accessible from population-level metabolomic data.
Journal ArticleDOI

Centrosome Separation and Central Spindle Assembly Act in Redundant Pathways that Regulate Microtubule Density and Trigger Cleavage Furrow Formation

Reinhard Dechant, +1 more
- 01 Mar 2003 - 
TL;DR: It is shown that in C. elegans embryos, although the central spindle is usually dispensable for furrow initiation, it becomes essential for fur row formation when the extent of centrosome separation during anaphase is reduced.
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Reversible protein aggregation is a protective mechanism to ensure cell cycle restart after stress

Shady Saad, +5 more
- 01 Oct 2017 - 
TL;DR: Stress-induced, reversible aggregation of yeast pyruvate kinase, Cdc19 is characterized and a region of low compositional complexity (LCR) within CDC19 is identified as necessary and sufficient for reversible aggregation.