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Hal E. Broxmeyer

Researcher at Indiana University

Publications -  616
Citations -  39797

Hal E. Broxmeyer is an academic researcher from Indiana University. The author has contributed to research in topics: Progenitor cell & Haematopoiesis. The author has an hindex of 99, co-authored 602 publications receiving 37839 citations. Previous affiliations of Hal E. Broxmeyer include University of Paris & University of Milan.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Hematopoietic reconstitution in a patient with fanconi's anemia by means of umbilical-cord blood from an hla-identical sibling

Eliane Gluckman, +14 more
- 26 Oct 1989 - 
TL;DR: It is necessary to select patients suitable for vaginal or laparoscopic mesh placement for Fanconi's anemia preoperatively on the basis of prior history and once they provide informed consent for surgery.
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Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice.

Landin Boring, +6 more
- 15 Nov 1997 - 
TL;DR: It is concluded that CCR2-/- mice have significant defects in both delayed-type hypersensitivity responses and production of Th1-type cytokines, suggesting an important and unexpected role for C CR2 activation in modulating the immune response, as well as in recruiting monocytes/macrophages to sites of inflammation.
Journal Article

The suppressive influences of human tumor necrosis factors on bone marrow hematopoietic progenitor cells from normal donors and patients with leukemia: synergism of tumor necrosis factor and interferon-gamma.

Hal E. Broxmeyer, +7 more
- 15 Jun 1986 - 
TL;DR: The suppressive effects of TNF did not appear to be mediated via endogenous T lymphocytes and/or monocytes in the bone marrow preparation, and a pulse exposure of marrow cells with TNF for 60 min resulted in maximal or near maximal inhibition when compared with cells left with T NF for the full culture incubation period.
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Cutting edge: IL-17F, a novel cytokine selectively expressed in activated T cells and monocytes, regulates angiogenesis and endothelial cell cytokine production.

Trevor Starnes, +6 more
- 15 Oct 2001 - 
TL;DR: Recombinant human IL-17F did not stimulate the proliferation of hematopoietic progenitors or the migration of mature leukocytes, however, it markedly inhibited the angiogenesis of human endothelial cells and induced endotocyte cells to produce IL-2, TGF-β, and monocyte chemoattractant protein-1.