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Jeroen J.M. Hoozemans

Researcher at University of Amsterdam

Publications -  171
Citations -  15066

Jeroen J.M. Hoozemans is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Unfolded protein response & Medicine. The author has an hindex of 52, co-authored 139 publications receiving 13224 citations. Previous affiliations of Jeroen J.M. Hoozemans include VU University Amsterdam & Leipzig University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The Unfolded Protein Response Is Activated in Pretangle Neurons in Alzheimer’s Disease Hippocampus

Jeroen J.M. Hoozemans, +6 more
- 01 Apr 2009 - 
TL;DR: Data indicate that UPR activation in AD neurons occurs at an early stage of neurofibrillary degeneration and suggest that the prolonged activation of the UPR is involved in both tau phosphorylation and neurodegeneration in AD pathogenesis.
Journal ArticleDOI

The unfolded protein response is activated in Alzheimer's disease.

Jeroen J.M. Hoozemans, +7 more
- 23 Jun 2005 - 
TL;DR: Data show that the UPR is activated in AD, and the increased occurrence of BiP/GRP78 and p-PERK in cytologically normal-appearing neurons suggest a role for the U PR early in AD neurodegeneration.
Journal ArticleDOI

Activation of the unfolded protein response in Parkinson's disease

Jeroen J.M. Hoozemans, +5 more
- 16 Mar 2007 - 
TL;DR: In this article, the authors investigated UPR activation in the substantia nigra of control and PD patients and found that the UPR is activated in PD and associated with the accumulation and aggregation of alpha-synuclein.
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Nrf2-induced antioxidant protection : A promising target to counteract ROS-mediated damage in neurodegenerative disease?

Helga E. de Vries, +6 more
- 15 Nov 2008 - 
TL;DR: An overview of the involvement of ROS-induced oxidative damage in Alzheimer's disease, Parkinson’s disease, and Huntington's disease is provided and the potential therapeutic effects of antioxidant enzymes and compounds that activate the Nrf2-ARE pathway are discussed.