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Sheng Li

Researcher at South China Normal University

Publications -  203
Citations -  10934

Sheng Li is an academic researcher from South China Normal University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 37, co-authored 127 publications receiving 8322 citations. Previous affiliations of Sheng Li include Johns Hopkins University School of Medicine & Chinese Academy of Sciences.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Fat Body Biology in the Last Decade.

Sheng Li, +2 more
- 07 Jan 2019 - 
TL;DR: The insect fat body is analogous to vertebrate adipose tissue and liver and has emerged as a fascinating model for studying metabolic disorders and immune diseases.
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Improvement of Pest Resistance in Transgenic Tobacco Plants Expressing dsRNA of an Insect-Associated Gene EcR

Jin-Qi Zhu, +10 more
- 07 Jun 2012 - 
TL;DR: This study reports that pest resistance is improved in transgenic tobacco plants expressing dsRNA of EcR from the cotton bollworm, Helicoverpa armigera, a serious lepidopteran pest for a variety of crops.
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Drosophila Met and Gce are partially redundant in transducing juvenile hormone action

Mohamed A. Abdou, +10 more
- 01 Dec 2011 - 
TL;DR: The data suggest a model in which Drosophila Met and Gce redundantly transduce JH action to prevent 20E-induced caspase-dependent PCD during larval molts by induction of Kr-h1 expression and inhibition of br expression.