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Edward A. Ratovitski

Researcher at Johns Hopkins University

Publications -  84
Citations -  11356

Edward A. Ratovitski is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Transcription factor & Cancer. The author has an hindex of 40, co-authored 84 publications receiving 9693 citations. Previous affiliations of Edward A. Ratovitski include Johns Hopkins University School of Medicine & George Washington University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

AIS is an oncogene amplified in squamous cell carcinoma

TL;DR: The results support the idea that AIS plays an oncogenic role in human cancer, and include evidence that p40/p73L, two variants lacking the N-terminal transactivation domain, in cancer.
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Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: The challenge ahead

William H. Goodson, +180 more
- 01 Jun 2015 - 
TL;DR: Low-dose exposures to common environmental chemicals that are deemed safe individually may be combining to instigate carcinogenesis, thereby contributing to the incidence of cancer.
Journal Article

ΔNp63α and TAp63α regulate transcription of genes with distinct biological functions in cancer and development

TL;DR: The p63 gene shows remarkable structural similarity to the p53 and p73 genes, and a survey of these genes shows that p63 can regulate a wide range of downstream gene targets with various cellular functions, including cell cycle control, stress, and signal transduction.