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Alina Maloyan

Researcher at Oregon Health & Science University

Publications -  57
Citations -  8945

Alina Maloyan is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Placenta & Medicine. The author has an hindex of 26, co-authored 49 publications receiving 7310 citations. Previous affiliations of Alina Maloyan include Hebrew University of Jerusalem & Cardiovascular Institute of the South.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Desmin-related cardiomyopathy in transgenic mice: a cardiac amyloidosis.

TL;DR: It is shown that expression of CryAB(R120G) leads to the formation of electron-dense bodies characteristic of the DRMs and identify these bodies as aggresomes, which are characteristic ofThe neurodegenerative diseases.
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MIR-210 modulates mitochondrial respiration in placenta with preeclampsia

TL;DR: The data collectively suggest that miR-210 overexpression during PE could be responsible for placental mitochondria dysfunction, and miRNAs tested showed significant up regulation in PE and significant downregulation of its target, ISCU mRNA.
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Mitochondrial Dysfunction and Apoptosis Underlie the Pathogenic Process in α-B-Crystallin Desmin-Related Cardiomyopathy

TL;DR: Cardiac chaperone dysfunction acutely leads to altered cardiomyocyte mechanics, perturbations in mitochondrial-sarcomere architecture, and deficits in mitochondrial function, which can result in activation of apoptosis and heart failure.