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Steve Lancel

Researcher at Pasteur Institute

Publications -  73
Citations -  9046

Steve Lancel is an academic researcher from Pasteur Institute. The author has contributed to research in topics: Mitochondrion & Mitochondrial biogenesis. The author has an hindex of 32, co-authored 66 publications receiving 7837 citations. Previous affiliations of Steve Lancel include French Institute of Health and Medical Research & university of lille.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Rev-erb-α modulates skeletal muscle oxidative capacity by regulating mitochondrial biogenesis and autophagy.

Estelle Woldt, +17 more
- 01 Aug 2013 - 
TL;DR: It is shown that Rev-erb-α is highly expressed in oxidative skeletal muscle and that its deficiency in muscle leads to reduced mitochondrial content and oxidative function, as well as upregulation of autophagy, which results in compromised exercise capacity.
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Targeting Oxidative Stress and Mitochondrial Dysfunction in the Treatment of Impaired Wound Healing: A Systematic Review.

Mariola Cano Sanchez, +4 more
- 24 Jul 2018 - 
TL;DR: Among available antioxidant strategies, treatments using mitochondrial-targeted antioxidants are of particular interest and have the potential to mitigate mitochondrial dysfunction and aberrant inflammatory response through activation of nucleotide-binding oligomerization domain (NOD)-like family receptors.
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Mitochondrial proliferation during apoptosis induced by anticancer agents: effects of doxorubicin and mitoxantrone on cancer and cardiac cells.

Jerome Kluza, +7 more
- 16 Sep 2004 - 
TL;DR: The proliferation of mitochondria could explain the higher toxicity of doxorubicin to cancer cells compared to cardiac cells and this suggests novel therapeutic opportunities to better control the cardiotoxicity of anthracyclines.
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Carbon Monoxide Rescues Mice from Lethal Sepsis by Supporting Mitochondrial Energetic Metabolism and Activating Mitochondrial Biogenesis

Steve Lancel, +5 more
- 01 May 2009 - 
TL;DR: Results reveal that delivery of controlled amounts of CO dramatically reduced mortality in septic mice, indicating that CO-RMs could be used therapeutically to prevent organ dysfunction and death in sepsis.