scispace - formally typeset
F

Florian Gruber

Researcher at Medical University of Vienna

Publications -  116
Citations -  11363

Florian Gruber is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Autophagy & Senescence. The author has an hindex of 37, co-authored 93 publications receiving 9363 citations. Previous affiliations of Florian Gruber include University of Vienna & University of Natural Resources and Life Sciences, Vienna.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Identification of a Novel Macrophage Phenotype That Develops in Response to Atherogenic Phospholipids via Nrf2

Alexandra Kadl, +14 more
- 17 Sep 2010 - 
TL;DR: The unique biological properties of Mox macrophages suggest this phenotype may play an important role in atherosclerotic lesion development as well as in other settings of chronic inflammation.
Journal ArticleDOI

Protective role of phospholipid oxidation products in endotoxin-induced tissue damage

Valery N. Bochkov, +5 more
- 05 Sep 2002 - 
TL;DR: It is shown that oxidized phospholipids inhibit LPS-induced but not tumour-necrosis factor-α-induced or interleukin-1β-induced NFκB-mediated upregulation of inflammatory genes, by blocking the interaction of LPS with L PS-binding protein and CD14.
Journal ArticleDOI

Knockdown of Filaggrin Impairs Diffusion Barrier Function and Increases UV Sensitivity in a Human Skin Model

Michael Mildner, +13 more
- 01 Sep 2010 - 
TL;DR: It is demonstrated that knockdown of filaggrin expression in an organotypic skin model reproduces epidermal alterations caused by filag Grin mutations in vivo, and a link between filag grin and endogenous UVB protection is established.