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Peter Hoet

Researcher at Katholieke Universiteit Leuven

Publications -  266
Citations -  18995

Peter Hoet is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: DNA methylation & Bronchoalveolar lavage. The author has an hindex of 61, co-authored 248 publications receiving 16193 citations. Previous affiliations of Peter Hoet include The Catholic University of America & Catholic University of Leuven.

Papers
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Journal ArticleDOI

Passage of inhaled particles into the blood circulation in humans.

Abderrahim Nemmar, +8 more
- 29 Jan 2002 - 
TL;DR: It is concluded that inhaled 99mTc-labeled ultrafine carbon particles pass rapidly into the systemic circulation, and this process could account for the well-established, but poorly understood, extrapulmonary effects of air pollution.
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Nanoparticles – known and unknown health risks

Peter Hoet, +2 more
- 08 Dec 2004 - 
TL;DR: This review provides comprehensive analysis of data available on health effects of nanomaterials and predicts a further rise in consumer products relying on nanotechnology.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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The nanosilica hazard: another variable entity

Dorota Napierska, +4 more
- 03 Dec 2010 - 
TL;DR: The physico-chemical properties of the different nano-sized silica materials that can affect their interaction with biological systems are summarized, with a specific emphasis on inhalation exposure.
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Loss of HIF-2α and inhibition of VEGF impair fetal lung maturation, whereas treatment with VEGF prevents fatal respiratory distress in premature mice

Veerle Compernolle, +16 more
- 10 Jun 2002 - 
TL;DR: It is reported that loss of the hypoxia-inducible transcription factor-2 α (HIF-2α) caused fatal RDS in neonatal mice due to insufficient surfactant production by alveolar type 2 cells and the pneumotrophic effect of VEGF may have therapeutic potential for lung maturation in preterm infants.