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Jihoon Nah

Researcher at Rutgers University

Publications -  38
Citations -  2993

Jihoon Nah is an academic researcher from Rutgers University. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 18, co-authored 29 publications receiving 1614 citations. Previous affiliations of Jihoon Nah include Seoul National University & University of Medicine and Dentistry of New Jersey.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Overexpression of Atg5 in mice activates autophagy and extends lifespan

Jong Ok Pyo, +7 more
- 13 Aug 2013 - 
TL;DR: It is shown that ubiquitous overexpression of Atg5, a protein essential for autophagosome formation, extends median lifespan of mice by 17.2%, and that Atg 5 transgenic mice showed anti-ageing phenotypes, including leanness, increased insulin sensitivity and improved motor function.
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Molecules and their functions in autophagy

Jong Ok Pyo, +2 more
- 29 Feb 2012 - 
TL;DR: The recent process of autophagy is summarized with emphasis on molecules and their protein complexes along with advanced molecular mechanisms that regulate the autophagic machinery.
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Autophagy in Neurodegenerative Diseases: From Mechanism to Therapeutic Approach

Jihoon Nah, +2 more
- 31 May 2015 - 
TL;DR: The overall role of autophagy in neurodegeneration is described, focusing on various therapeutic strategies for modulating specific stages of autophile and on the current status of drug development.
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An alternative mitophagy pathway mediated by Rab9 protects the heart against ischemia

Toshiro Saito, +18 more
- 01 Feb 2019 - 
TL;DR: It is shown that mitophagy during myocardial ischemia was mediated predominantly through autophagy characterized by Rab9-associated autophagosomes, rather than the well-characterized form of autophagic that is dependent on the autophile-related 7 (Atg) conjugation system and LC3.