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Anne-Claire Jacomin

Researcher at University of Warwick

Publications -  25
Citations -  1651

Anne-Claire Jacomin is an academic researcher from University of Warwick. The author has contributed to research in topics: Autophagy & Transcription factor. The author has an hindex of 13, co-authored 23 publications receiving 653 citations. Previous affiliations of Anne-Claire Jacomin include University of Grenoble & Joseph Fourier University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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iLIR database: A web resource for LIR motif-containing proteins in eukaryotes.

TL;DR: The iLIR database is developed, listing all the putative canonical LIRCPs identified in silico in the proteomes of 8 model organisms using the iL IR server, and a curated text-mining analysis of the literature permitted us to identify novel putative LICRPs in mammals that have not been associated with autophagy.
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The deubiquitinating enzyme USP36 controls selective autophagy activation by ubiquitinated proteins.

TL;DR: It is reported here that the deubiquitinating enzyme USP36 controls selective autophagy activation in Drosophila and in human cells, and it is shown that dUsp36 loss of function autonomously inhibits cell growth while activating Autophagy.
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Identification of a plant gene encoding glutamate/aspartate-prephenate aminotransferase: the last homeless enzyme of aromatic amino acids biosynthesis.

TL;DR: The data revealed that this activity is housed by the prokaryotic‐type plastidic aspartate aminotransferase (At2g22250), which represents the first identification of a gene encoding PAT.
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Targeted Interplay Between Bacterial Pathogens and Host Autophagy

TL;DR: This analysis revealed that autophagy receptors in general potentially target mostly genus-specific proteins, and not those present in multiple genera, and observed apathogen-specific pattern as to which autophagic phase could be modulated by specific genera.