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Gary Sweeney

Researcher at York University

Publications -  155
Citations -  10624

Gary Sweeney is an academic researcher from York University. The author has contributed to research in topics: Adiponectin & Glucose uptake. The author has an hindex of 54, co-authored 143 publications receiving 8736 citations. Previous affiliations of Gary Sweeney include University of British Columbia & University of Hong Kong.

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Cardiac Remodeling in Obesity

TL;DR: The role of leptin and the emerging promise of adiponectin as a cardioprotective agent are focused on and the ways in which obesity can influence structure and function of the heart are summarized.
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Lipocalin-2 Is an Inflammatory Marker Closely Associated with Obesity, Insulin Resistance, and Hyperglycemia in Humans

TL;DR: Lipocalin-2 is an inflammatory marker closely related to obesity and its metabolic complications and might be useful for evaluating the outcomes of various clinical interventions for obesity-related metabolic and cardiovascular diseases.
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Globular adiponectin increases GLUT4 translocation and glucose uptake but reduces glycogen synthesis in rat skeletal muscle cells.

TL;DR: The present study is the first to show that globular adiponectin increases glucose uptake in skeletal muscle cells via GLUT4 translocation and subsequently reduces the rate of glycogen synthesis and shifts glucose metabolism toward lactate production.
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Analysis of paradoxical observations on the association between leptin and insulin resistance

TL;DR: A summary of paradoxical observations on the effects of leptin on glucose homeostasis and the ability of leptin to induce or improve insulin resistance suggests that a complex interplay exists between direct peripheral and centrally mediated effects of the hormone.