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Eric Ogier-Denis

Researcher at University of Paris

Publications -  108
Citations -  7916

Eric Ogier-Denis is an academic researcher from University of Paris. The author has contributed to research in topics: Cellular differentiation & Inflammatory bowel disease. The author has an hindex of 37, co-authored 99 publications receiving 6665 citations. Previous affiliations of Eric Ogier-Denis include Beaujon Hospital & University of Rennes.

Papers
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Journal ArticleDOI

Distinct Classes of Phosphatidylinositol 3′-Kinases Are Involved in Signaling Pathways That Control Macroautophagy in HT-29 Cells

Anne Petiot, +4 more
- 14 Jan 2000 - 
TL;DR: The results demonstrate that distinct classes of PI3K control the macroautophagic pathway in opposite directions.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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The Tumor Suppressor PTEN Positively Regulates Macroautophagy by Inhibiting the Phosphatidylinositol 3-Kinase/Protein Kinase B Pathway

Sébastien Arico, +6 more
- 21 Sep 2001 - 
TL;DR: The data demonstrate that the signaling control of macroautophagy overlaps with the well known PI 3-kinase/PKB survival pathway and that the loss of PTEN function in cancer cells inhibits a major catabolic pathway.
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NAD(P)H Oxidase Nox-4 Mediates 7-Ketocholesterol-Induced Endoplasmic Reticulum Stress and Apoptosis in Human Aortic Smooth Muscle Cells

Eric Pedruzzi, +15 more
- 15 Dec 2004 - 
TL;DR: It is shown that 7-ketocholesterol (7-Kchol) induces oxidative stress and/or apoptotic events in human aortic smooth muscle cells (SMCs), which is consistent with the hypothesis that Nox-4/oxysterols are involved in the pathogenesis of atherosclerosis.
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Autophagy: a barrier or an adaptive response to cancer.

Eric Ogier-Denis, +1 more
- 17 Mar 2003 - 
TL;DR: Autophagy as a stress response mechanism protects cancer cells from low nutrient supply or therapeutic insults and is also involved in the elimination of cancer cells by triggering a non-apoptotic cell death program, suggesting a negative role in tumor development.