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Rohan Dhiman

Researcher at National Institute of Technology, Rourkela

Publications -  60
Citations -  2593

Rohan Dhiman is an academic researcher from National Institute of Technology, Rourkela. The author has contributed to research in topics: Mycobacterium tuberculosis & Autophagy. The author has an hindex of 16, co-authored 51 publications receiving 1223 citations. Previous affiliations of Rohan Dhiman include University of Texas Health Science Center at San Antonio & Council of Scientific and Industrial Research.

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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One pot synthesis of CdS/BiOBr/Bi2O2CO3: A novel ternary double Z-scheme heterostructure photocatalyst for efficient degradation of atrazine

TL;DR: In this article, a simple one-step hydrothermal method was developed for morphology controlled synthesis of CdS/BiOBr/Bi2O2CO3 ternary heterostructure materials.
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IL-22 Produced by Human NK Cells Inhibits Growth of Mycobacterium tuberculosis by Enhancing Phagolysosomal Fusion

TL;DR: It is concluded that NK cells can contribute to immune defenses against M. tuberculosis through production of IL-22, which inhibits intracellular mycobacterial growth by enhancing phagolysosomal fusion.
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Structure-function and application of plant lectins in disease biology and immunity.

TL;DR: It is found that many plant lectins mediate its microbicidal activity by triggering host immune responses that result in the release of several cytokines followed by activation of effector mechanism.
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Programmed death 1 and cytokine inducible SH2-containing protein dependent expansion of regulatory T cells upon stimulation With Mycobacterium tuberculosis.

TL;DR: M. tuberculosis infection induces development of Tregs from CCR4(+) cells through a process that depends on PD-1and CISH, which is found to be dependent on programmed death1 and CISH.