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Grzegorz Węgrzyn

Researcher at University of Gdańsk

Publications -  458
Citations -  11688

Grzegorz Węgrzyn is an academic researcher from University of Gdańsk. The author has contributed to research in topics: Escherichia coli & Gene. The author has an hindex of 47, co-authored 413 publications receiving 9187 citations. Previous affiliations of Grzegorz Węgrzyn include Polish Academy of Sciences & Laboratory of Molecular Biology.

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Staphylococcus aureus as an infectious agent: overview of biochemistry and molecular genetics of its pathogenicity.

TL;DR: The biochemical and genetic mechanisms of pathogenicity of S. aureus strains, Virulence factors, organization of the genome and regulation of expression of genes involved in virulence, and mechanisms leading to methicilin resistance are presented and briefly discussed.
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Biodiversity of bacteriophages: morphological and biological properties of a large group of phages isolated from urban sewage.

TL;DR: In this article, a collection of 83 bacteriophages, isolated from urban sewage and able to propagate in cells of different bacterial hosts, has been obtained (60 infecting Escherichia coli, 10 infecting Pseudomonas aeruginosa, 4 infecting Salmonella enterica, 3 infecting Staphylococcus sciuri, and 6 infecting Enterococcus faecalis).
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Genistein-mediated inhibition of glycosaminoglycan synthesis as a basis for gene expression-targeted isoflavone therapy for mucopolysaccharidoses

TL;DR: It is found that genistein inhibits synthesis of GAGs considerably in cultures of fibroblasts of MPS patients and is proposed to consider a substrate reduction therapy for MPS, which is referred to as ‘gene expression-targeted isoflavone therapy’.