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Xingguo Liu

Researcher at Guangzhou Institutes of Biomedicine and Health

Publications -  84
Citations -  2956

Xingguo Liu is an academic researcher from Guangzhou Institutes of Biomedicine and Health. The author has contributed to research in topics: Mitochondrion & Reprogramming. The author has an hindex of 19, co-authored 66 publications receiving 1564 citations. Previous affiliations of Xingguo Liu include University of Science and Technology of China & Tsinghua University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Mitochondrial 'kiss-and-run': interplay between mitochondrial motility and fusion-fission dynamics.

Xingguo Liu, +3 more
- 21 Oct 2009 - 
TL;DR: Opa1 expression and cytoskeletal anchorage govern a novel form of fusion that has a distinct function in mitochondrial maintenance and both required and promoted mitochondrial motility.
Journal ArticleDOI

Altered fusion dynamics underlie unique morphological changes in mitochondria during hypoxia–reoxygenation stress

Xingguo Liu, +1 more
- 04 Mar 2011 - 
TL;DR: The metabolic stress during hypoxia–reoxygenation alters mitochondrial morphology by inducing distinct patterns of mitochondrial dynamics, which includes processes that could aid mitochondrial adaptation and functional recovery.
Journal ArticleDOI

Ca2+-dependent regulation of mitochondrial dynamics by the Miro-Milton complex.

Xingguo Liu, +1 more
- 01 Oct 2009 - 
TL;DR: Evidence is emerging that calcium signaling through Miro-Milton is central to coordination of the local oxidative metabolism with the energy demands and protection against Ca(2+)-induced cell injury.
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Valproic acid-induced hepatotoxicity in Alpers syndrome is associated with mitochondrial permeability transition pore opening-dependent apoptotic sensitivity in an induced pluripotent stem cell model.

Shengbiao Li, +9 more
- 01 May 2015 - 
TL;DR: AHS iPSCs‐Hep are more sensitive to the VPA‐induced mitochondrial‐dependent apoptotic pathway, and this effect is mediated by mPTP opening, suggesting that targeting mP TP opening could be an effective method to prevent hepatotoxicity by VPA in AHS patients.