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Giacinto Bagetta

Researcher at University of Calabria

Publications -  271
Citations -  10125

Giacinto Bagetta is an academic researcher from University of Calabria. The author has contributed to research in topics: Neuroprotection & Medicine. The author has an hindex of 48, co-authored 242 publications receiving 7978 citations. Previous affiliations of Giacinto Bagetta include University of Messina & University of Cagliari.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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CXCR4-activated astrocyte glutamate release via TNFalpha: amplification by microglia triggers neurotoxicity.

Paola Bezzi, +10 more
- 01 Jul 2001 - 
TL;DR: It is demonstrated that altered glial communication has direct neuropathological consequences and that agents interfering with CXCR4-dependent astrocyte–microglia signaling prevent neuronal apoptosis induced by the HIV-1 coat glycoprotein, gp120IIIB.
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Post‐ischemic brain damage: pathophysiology and role of inflammatory mediators

Diana Amantea, +4 more
- 01 Jan 2009 - 
TL;DR: The present work reviewed the role of neuroinflammatory mediators in the pathophysiology of ischemic brain damage and their potential exploitation as drug targets for the treatment of cerebral ischemia.
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From clinical evidence to molecular mechanisms underlying neuroprotection afforded by estrogens.

Diana Amantea, +3 more
- 01 Aug 2005 - 
TL;DR: Recent findings concerning the neuronal effects of estrogens that may contribute to their neuroprotective actions are discussed, and both estrogen receptor-dependent and -independent mechanisms will be described.
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Rational modulation of the innate immune system for neuroprotection in ischemic stroke.

Diana Amantea, +8 more
- 29 Apr 2015 - 
TL;DR: Immunity cells exert a dualistic role on the evolution of ischemic brain damage, since the classic phenotypes promote injury, whereas alternatively activated M2 macrophages or N2 neutrophils prompt tissue remodeling and repair.