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William T. Jackson

Researcher at University of Maryland, Baltimore

Publications -  50
Citations -  16134

William T. Jackson is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: Autophagy & Viral replication. The author has an hindex of 26, co-authored 45 publications receiving 13499 citations. Previous affiliations of William T. Jackson include Stanford University & Medical College of Wisconsin.

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Topology of Double-Membraned Vesicles and the Opportunity for Non-Lytic Release of Cytoplasm

TL;DR: It is hypothesize that autophagosome-like double-membranes, which can become single- Membranes upon maturation, provide a long-sought mechanism for the observed non-lytic release of cytoplasmic viruses and possibly other cytopLasmic material resistant to the environment of maturing autophosomes.
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Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
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Poliovirus induces autophagic signaling independent of the ULK1 complex.

TL;DR: The data suggest that PV has a finely-tuned relationship with the autophagic machinery, generating autophagosomes without using the primary autophagy signaling pathway.
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Fragmentation of the Golgi apparatus provides replication membranes for human rhinovirus 1A.

TL;DR: Data show that in HRV-1A infection, the 3A protein plays a role in fragmenting the Golgi complex and generating vesicles that are used as the site of viral RNA replication.
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BPIFB3 Regulates Autophagy and Coxsackievirus B Replication through a Noncanonical Pathway Independent of the Core Initiation Machinery

TL;DR: A previously uncharacterized regulator of enterovirus infection that controls replication through a noncanonical pathway independent from the core autophagy initiation machinery is reported on.