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Hermann Pavenstädt

Researcher at University of Münster

Publications -  91
Citations -  9097

Hermann Pavenstädt is an academic researcher from University of Münster. The author has contributed to research in topics: Transplantation & Kidney. The author has an hindex of 29, co-authored 86 publications receiving 7988 citations. Previous affiliations of Hermann Pavenstädt include Heidelberg University & University of Freiburg.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice

Markus Gödel, +23 more
- 01 Jun 2011 - 
TL;DR: It is suggested that mTOR inhibition can protect podocytes and prevent progressive diabetic nephropathy and demonstrate the requirement for tightly balanced mTOR activity in podocyte homeostasis.
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Cisplatin Nephrotoxicity Is Critically Mediated via the Human Organic Cation Transporter 2

Giuliano Ciarimboli, +9 more
- 01 Dec 2005 - 
TL;DR: It is demonstrated that uptake of cis-platin is mediated by hOCT2 in renal proximal tubules, explaining its organ-specific toxicity.
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Developmental signalling pathways in renal fibrosis: the roles of Notch, Wnt and Hedgehog

Maria Edeling, +4 more
- 01 Jul 2016 - 
TL;DR: This work describes how three key developmental signalling pathways — Notch, Wnt and Hedgehog (Hh) — are reactivated in response to kidney injury and contribute to the fibrotic response.
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Increased inorganic phosphate induces human endothelial cell apoptosis in vitro

G. S. Di Marco, +6 more
- 01 Jun 2008 - 
TL;DR: Findings show that hyperphosphatemia causes endothelial cell apoptosis, a process that impairs endothelial integrity, and may be an initial event leading to vascular complications in patients with chronic kidney disease.