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Angelika Amon

Researcher at Massachusetts Institute of Technology

Publications -  201
Citations -  29098

Angelika Amon is an academic researcher from Massachusetts Institute of Technology. The author has contributed to research in topics: Aneuploidy & Mitotic exit. The author has an hindex of 78, co-authored 200 publications receiving 26171 citations. Previous affiliations of Angelika Amon include Cold Spring Harbor Laboratory & University of California, San Francisco.

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Journal ArticleDOI

Aneuploid proliferation defects in yeast are not driven by copy number changes of a few dosage-sensitive genes

Megan E. Bonney, +2 more
- 01 May 2015 - 
TL;DR: It is concluded that phenotypic thresholds can be crossed by mass action of copy number changes that, on their own, are benign.
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Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
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Phosphorylation-Mediated Clearance of Amyloid-like Assemblies in Meiosis.

Kayla Carpenter, +4 more
- 07 May 2018 - 
TL;DR: It is shown that multi-site phosphorylation of Rim4 is critical for its regulated disassembly and degradation and that failure to clear Rim4 assemblies interferes with meiotic progression, and that the natural pathways by which cells process these structures could be deployed to act on disease-related amyloids.
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Cell size is a determinant of stem cell potential during aging

Jette Lengefeld, +21 more
- 12 Nov 2021 - 
TL;DR: This paper found that hematopoietic stem cells (HSCs) enlarge under conditions known to decrease stem cell function and that this decreased fitness of large HSCs is due to reduced proliferation and was accompanied by altered metabolism.
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Why haploinsufficiency persists.

Summer A Morrill, +1 more
- 11 Jun 2019 - 
TL;DR: The origin of haploinsufficiency is investigated by creating a comprehensive dosage sensitivity data set for genes under their native promoters, and a dosage-stabilizing hypothesis is proposed to explain its persistence over evolutionary time.