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Xiaoyan Jiang

Researcher at Tongji University

Publications -  209
Citations -  11393

Xiaoyan Jiang is an academic researcher from Tongji University. The author has contributed to research in topics: Myeloid leukemia & Stem cell. The author has an hindex of 40, co-authored 186 publications receiving 10162 citations. Previous affiliations of Xiaoyan Jiang include BC Cancer Agency & Harbin Medical University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Isolation of a highly quiescent subpopulation of primitive leukemic cells in chronic myeloid leukemia.

Tessa L. Holyoake, +3 more
- 15 Sep 1999 - 
TL;DR: These findings provide the first direct and definitive evidence of a deeply but reversibly quiescent subpopulation of leukemic cells in patients with CML with both in vitro and in vivo stem cell properties.
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Chronic myeloid leukemia stem cells possess multiple unique features of resistance to BCR-ABL targeted therapies.

Xiaoyan Jiang, +9 more
- 01 May 2007 - 
TL;DR: It is shown that the lesser effect of imatinib mesylate on the 3-week output of cells produced in vitro from lin−CD34+CD38− CML (stem) cells compared with cultures initiated with the CD38+ subset of lin− CD34+ cells is markedly enhanced when conditions of reduced growth factor stimulation are used.
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Autocrine production and action of IL-3 and granulocyte colony-stimulating factor in chronic myeloid leukemia

Xiaoyan Jiang, +4 more
- 26 Oct 1999 - 
TL;DR: It is shown from analyses of growth-factor gene expression, protein production, and antibody inhibition studies that this deregulated growth can be explained, at least in part, by a novel differentiation-controlled autocrine mechanism.
Journal ArticleDOI

Stable Suppression of a Novel Oncogene, AHI-1, in Human Cutaneous T-Cell Leukemia Cells Normalizes Its Transforming Activity In Vitro and In Vivo and Aberrant Expression of AHI-1 Is Also Present in Leukemic Sezary Cells from Patients with Sezary Syndrome.

Ashley Ringrose, +7 more
- 16 Nov 2005 - 
TL;DR: Findings provide strong evidence of the oncogenic activity of AHI-1 in human T-cell leukemic cells and its deregulation can contribute to the development of human cutaneous T- cell lymphomas, including Sezary syndrome.